CSNpharm
1-Deoxynojirimycin is a potent α-glucosidase inhibitor, suppresses postprandial blood glucose, thereby possibly preventing diabetes mellitus. It’s a natural product isolated and purified from the root barks of Morus alba L..
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CSNpharm
1-Deoxynojirimycin is a potent α-glucosidase inhibitor, suppresses postprandial blood glucose, thereby possibly preventing diabetes mellitus. It’s a natural product isolated and purified from the root barks of Morus alba L..
More Information
Supplier Page
CSNpharm
1-Deoxynojirimycin is a potent α-glucosidase inhibitor, suppresses postprandial blood glucose, thereby possibly preventing diabetes mellitus. It’s a natural product isolated and purified from the root barks of Morus alba L..
More Information
Supplier Page
CSNpharm
L-Chicoric acid, a natural product isolated and purified from the herbs of Echinacea purpurea, is an inhibitor of human immunodeficiency virus type 1 (HIV-1) integrase in vitro and of HIV-1 replication in tissue culture.
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CSNpharm
L-Chicoric acid, a natural product isolated and purified from the herbs of Echinacea purpurea, is an inhibitor of human immunodeficiency virus type 1 (HIV-1) integrase in vitro and of HIV-1 replication in tissue culture.
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CSNpharm
L-Chicoric acid, a natural product isolated and purified from the herbs of Echinacea purpurea, is an inhibitor of human immunodeficiency virus type 1 (HIV-1) integrase in vitro and of HIV-1 replication in tissue culture.
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CSNpharm
Salicylic acid, a natural product extract from Willow bark, well known as an antiinflammatory inhibitor of cyclooxygenase activity.
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CSNpharm
Salicylic acid, a natural product extract from Willow bark, well known as an antiinflammatory inhibitor of cyclooxygenase activity.
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CSNpharm
Triclabendazole is a benzimidazole carbamate anthelmintic which can bind to beta-tublin and inhibits protein synthesis.
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CSNpharm
4-Hydroxybenzyl alcohol, a natural product isolated and purified from the herbs of Cistanche deserticola Ma, exhibits beneficial effects in cerebral ischemic injury, has neuroprotective effect through upregulation of Nrf2, Prdx6, and PDI expression via the PI3K/Akt pathway.
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