Search Results

URB602 10 mg  | 99.64%

TargetMol

URB602 is a specific monoacylglycerol lipase (MGL) inhibitor, which inhibits rat brain MGL (IC50: 28±4 μM) through a noncompetitive mechanism.

More Information Supplier Page

URB602 100 mg  | 99.64%

TargetMol

URB602 is a specific monoacylglycerol lipase (MGL) inhibitor, which inhibits rat brain MGL (IC50: 28±4 μM) through a noncompetitive mechanism.

More Information Supplier Page

KS176 5 mg  | 99.55%

TargetMol

KS176 is a potent and selective inhibitor of the breast cancer resistance protein (BCRP) multidrug transporter, showing no inhibitory activity against P-gp or MRP1.

More Information Supplier Page

KS176 50 mg  | 99.55%

TargetMol

KS176 is a potent and selective inhibitor of the breast cancer resistance protein (BCRP) multidrug transporter, showing no inhibitory activity against P-gp or MRP1.

More Information Supplier Page

NVP-ACC789 5 mg  | 99.19%

TargetMol

ACC-789 (NVP-ACC789; ZK-202650) is an effective, specific and orally active inhibitor of the VEGF receptor tyrosine kinases.

More Information Supplier Page

NVP-ACC789 50 mg  | 99.19%

TargetMol

ACC-789 (NVP-ACC789; ZK-202650) is an effective, specific and orally active inhibitor of the VEGF receptor tyrosine kinases.

More Information Supplier Page

NVP-ACC789 25 mg  | 99.19%

TargetMol

ACC-789 (NVP-ACC789; ZK-202650) is an effective, specific and orally active inhibitor of the VEGF receptor tyrosine kinases.

More Information Supplier Page

NVP-ACC789 10 mg  | 99.19%

TargetMol

ACC-789 (NVP-ACC789; ZK-202650) is an effective, specific and orally active inhibitor of the VEGF receptor tyrosine kinases.

More Information Supplier Page

S107 5 mg  | 99.63%

TargetMol

S107 is a RyR-selective 1, 4-benzothiazepine derivative that stabilizes RyR2 channels by enhancing the binding affinity of calstabin2 to mutant and/or PKA-phosphorylated channels.

More Information Supplier Page

S107 50 mg  | 99.63%

TargetMol

S107 is a RyR-selective 1, 4-benzothiazepine derivative that stabilizes RyR2 channels by enhancing the binding affinity of calstabin2 to mutant and/or PKA-phosphorylated channels.

More Information Supplier Page