Dihydropinosylvin, is a natural product isolated and purified from the herbs of Dioscorea rotundata with anti-fungal activity.

Dihydropinosylvin methyl ether is a natural product isolated and purified from the herbs of Pinus strobus with anti-bacterial effects.

Dihydroresveratrol, a potent phytoestrogen, is a hormone receptor modulator. Dihydroresveratrol exhibits proliferative effects in androgen-independent prostate and breast cancer cells at picomolar and nanomolar concentrations.

Dihydroresveratrol, a potent phytoestrogen, is a hormone receptor modulator. Dihydroresveratrol exhibits proliferative effects in androgen-independent prostate and breast cancer cells at picomolar and nanomolar concentrations.

Dihydroresveratrol, a potent phytoestrogen, is a hormone receptor modulator. Dihydroresveratrol exhibits proliferative effects in androgen-independent prostate and breast cancer cells at picomolar and nanomolar concentrations.

Dihydrotamarixetin is a natural product isolated and purified from the herbs of Heracleum stenopterum.

Dihydrotamarixetin is a natural product isolated and purified from the herbs of Heracleum stenopterum.

Dihydrotanshinone is extracted from the traditional chinese medicine salvia miltiorrhiza and is shown to inhibit several types of cancer.

Dihydrotanshinone is extracted from the traditional chinese medicine salvia miltiorrhiza and is shown to inhibit several types of cancer.

Dihydrotanshinone is extracted from the traditional chinese medicine salvia miltiorrhiza and is shown to inhibit several types of cancer.

Dihydrotanshinone is extracted from the traditional chinese medicine salvia miltiorrhiza and is shown to inhibit several types of cancer.

Dihydrotanshinone I, a natural product isolated and purified from the roots of Salvia miltiorrhiza Bge., exhibits dose-dependent inhibition towards HLM-catalyzed propofol glucuronidation. Dihydrotanshinone I showed inhibitory effect on PL(pro) with IC50 value of 4.9μM and less inhibitory effect on 3CLpro with IC50 value of 14.4μM.

Dihydrotanshinone I, a natural product isolated and purified from the roots of Salvia miltiorrhiza Bge., exhibits dose-dependent inhibition towards HLM-catalyzed propofol glucuronidation. Dihydrotanshinone I showed inhibitory effect on PL(pro) with IC50 value of 4.9μM and less inhibitory effect on 3CLpro with IC50 value of 14.4μM.

Dihydrotanshinone I, a natural product isolated and purified from the roots of Salvia miltiorrhiza Bge., exhibits dose-dependent inhibition towards HLM-catalyzed propofol glucuronidation. Dihydrotanshinone I showed inhibitory effect on PL(pro) with IC50 value of 4.9μM and less inhibitory effect on 3CLpro with IC50 value of 14.4μM.

Diloxanide furoat, a luminal amebicide used in the treatment of Amebiasis, is considered the luminal agent of choice for mild intestinal amebiasis.

DIM-C-pPhOCH3 is a Nur77 agonist. Nerve growth factor-induced Bα (NGFI-Bα, Nur77) is an orphan nuclear receptor.

DIM-C-pPhOCH3 is a Nur77 agonist. Nerve growth factor-induced Bα (NGFI-Bα, Nur77) is an orphan nuclear receptor.

DIM-C-pPhOCH3 is a Nur77 agonist. Nerve growth factor-induced Bα (NGFI-Bα, Nur77) is an orphan nuclear receptor.

DIM-C-pPhOCH3 is a Nur77 agonist. Nerve growth factor-induced Bα (NGFI-Bα, Nur77) is an orphan nuclear receptor.

DIMBOA is a maize derived growth inhibitor.

DIMBOA is a maize derived growth inhibitor.

DIMBOA is a maize derived growth inhibitor.

DIMBOA is a maize derived growth inhibitor.

Dimethylacrylshikonin, a natural product isolated and purified from the roots of Lithosperraum erythrorhizon Sieb. et Zucc. with anti-proliferate activity, is a promising agent for developing an improved strategy for radiotherapy against tumors.

Dimethylacrylshikonin, a natural product isolated and purified from the roots of Lithosperraum erythrorhizon Sieb. et Zucc. with anti-proliferate activity, is a promising agent for developing an improved strategy for radiotherapy against tumors.

ASC-J9, an antitumor agent, suppresses castration-resistant prostate cancer growth via degradation of full-length and splice variant androgen receptors.

ASC-J9, an antitumor agent, suppresses castration-resistant prostate cancer growth via degradation of full-length and splice variant androgen receptors.

ASC-J9, an antitumor agent, suppresses castration-resistant prostate cancer growth via degradation of full-length and splice variant androgen receptors.

ASC-J9, an antitumor agent, suppresses castration-resistant prostate cancer growth via degradation of full-length and splice variant androgen receptors.

ASC-J9, an antitumor agent, suppresses castration-resistant prostate cancer growth via degradation of full-length and splice variant androgen receptors.