β-Amyloid (12-20) is a peptide fragment of β-Amyloid.

β-amyloid (12-28) is a 17-aa peptide fragment, which can produce amyloid aggregates, used in the research of Alzheimer’s disease.

β-amyloid (15-21) is a fragment of Amyloid-β peptide, maybe used in the research of neurological disease.

β-Amyloid (18-28) is a peptide fragment of β-Amyloid.

β-Amyloid (22-35) is a 14-aa peptide, shows aggregates and induces neurotoxicity in the hippocampal cells.

β-Amyloid (22-40) is a peptide fragment of β-Amyloid.

β-Amyloid (29-40) is a fragment of Amyloid-β peptide.

β-Amyloid (31-35) is the shortest sequence of native Amyloid-β peptide that retains neurotoxic activity.

β-Amyloid (4-10) is an epitope for the polyclonal anti-Aβ(1-42) antibody, reduces amyloid deposition in a transgenic Alzheimer disease mouse model.

β-Aspartylaspartic acid is a natural compound found in Asparagus (Asparagus officinalis) Shoots.

The antihypertensive peptide isolated from fermented milk had a low inhibitory activity against angiotensin I-converting enzyme (ACE), IC50 720 μM. The dipeptide is a possible biosynthetic precursor of endomorphin.

β-Casomorphin (1-3), amide is a peptide fragment of β-Casomorphin with 3 amino acid.

β-Casomorphin (1-5), bovine is a peptide of bovine β-Casomorphin.

β-Casomorphin (1-5), amide, bovine is a peptide of bovine β-Casomorphin.

β-Casomorphin (1-6), bovine is a opioid-like bioactive peptide of β-Casomorphin.

β-casomorphin, bovine (β-casomorphin-7) is a opioid peptide with an IC50 of 14 μM in an Opioid receptors binding assay.

β-Casomorphin, human is an opioid peptide, acts as an agonist of opioid receptor.

β-catenin peptide is a 8-aa peptide, and can promote thymocyte positive selection.

Calcitonin Gene Related Peptide (CGRP) is a highly potent vasodialator and can function in the transmission of pain

β-CGRP, human TFA is one of calcitonin peptides, acts via the complex of calcitonin-receptor-like receptor (CRLR) and receptor-activity-modifying protein (RAMP), with IC50s of 1 nM and 300 nM for CRLR/RAMP1 and CRLR/RAMP2 in cells.

β-Endorphin is an endogenous opioid peptide that acts as an agonist at μ-opioid receptors (μORs) and exhibits immunomodulatory, neuromodulatory, antidepressant, and antinociceptive/analgesic activities. In vivo, β-endorphin increases levels of B-cells and production of antibodies as well as secretion of IL-4 and proliferation of splenocytes, shifting the immune response toward Th2-specific mediators.

β-Endorphin is an endogenous opioid peptide that acts as an agonist at μ-opioid receptors (μORs) and exhibits immunomodulatory, neuromodulatory, antidepressant, and antinociceptive/analgesic activities. In vivo, β-endorphin increases levels of B-cells and production of antibodies as well as secretion of IL-4 and proliferation of splenocytes, shifting the immune response toward Th2-specific mediators.

β-Endorphin, equine is an endogenous opioid peptide with analgesic properties.

β-Melanocyte Stimulating Hormone (MSH), human, a 22-residue peptide, acts as an endogenous melanocortin-4 receptor (MC4-R) agonist.

β-Melanocyte Stimulating Hormone (MSH), human TFA, a 22-residue peptide, acts as an endogenous melanocortin-4 receptor (MC4-R) agonist.

β-Nicotinamide Mononucleotide (β-NMN) is an intermediate in the biosynthesis of nicotinamide adenine dinucleotide (NAD+). Nicotinamide phosphoribosyltransferase (Nampt) catalyzes the condensation of nicotinamide with 5-phosphoribosyl-1-pyrophosphate to generate β-NMN, which is subsequently converted to NAD+ by β-NMN adenyltransferase. β-NMN has several beneficial pharmacological activities. Mostly mediated by its involvement in NAD+ biosynthesis, including its role in cellular […]