Adooq Bioscience

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AdooQ BioScience is a supplier of biochemicals located in Irvine, CA since 2006. In the past five years, we have been a renowned name engaged in manufacturing and supplying small molecule inhibitors including kinase inhibitors on kinds of signal pathway, such as PI3K, AKT, HDAC, MARP etc.

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11-oxo-mogroside V 25mg 25mg  | Purity Not Available

Adooq Bioscience

11-oxo-mogroside V showed a higher scavenging effect on O2- with EC50 of 4.79 mg/ml and H2O2 (EC50 of 16.52 mg/ ml) than those of mogroside V.

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11-oxo-mogroside V 5mg 5mg  | Purity Not Available

Adooq Bioscience

11-oxo-mogroside V showed a higher scavenging effect on O2- with EC50 of 4.79 mg/ml and H2O2 (EC50 of 16.52 mg/ ml) than those of mogroside V.

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12-O-tetradecanoyl phorbol-13-acetate 10mg 10mg  | Purity Not Available

Adooq Bioscience

12-O-tetradecanoyl phorbol-13-acetate is a diester of phorbol and is a tumor promoting compound extracted from croton oil. It is a reversible, highly potent protein kinase C (PKC) activator in vitro and in vivo at nM concentrations.

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12-O-tetradecanoyl phorbol-13-acetate 25mg 25mg  | Purity Not Available

Adooq Bioscience

12-O-tetradecanoyl phorbol-13-acetate is a diester of phorbol and is a tumor promoting compound extracted from croton oil. It is a reversible, highly potent protein kinase C (PKC) activator in vitro and in vivo at nM concentrations.

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12-O-tetradecanoyl phorbol-13-acetate 5mg 5mg  | Purity Not Available

Adooq Bioscience

12-O-tetradecanoyl phorbol-13-acetate is a diester of phorbol and is a tumor promoting compound extracted from croton oil. It is a reversible, highly potent protein kinase C (PKC) activator in vitro and in vivo at nM concentrations.

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1400W Dihydrochloride 10mg 10mg  | Purity Not Available

Adooq Bioscience

1400W Dihydrochloride is a slow, tight binding, potent and highly selective inhibitor of inducible nitric oxide synthase (Kd = 7 nM). Selective over nNOS and eNOS (Ki values are 2 and 50 ??M respectively). Cell-permeable and active in vivo.

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1400W Dihydrochloride 25mg 25mg  | Purity Not Available

Adooq Bioscience

1400W Dihydrochloride is a slow, tight binding, potent and highly selective inhibitor of inducible nitric oxide synthase (Kd = 7 nM). Selective over nNOS and eNOS (Ki values are 2 and 50 ??M respectively). Cell-permeable and active in vivo.

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1400W Dihydrochloride 50mg 50mg  | Purity Not Available

Adooq Bioscience

1400W Dihydrochloride is a slow, tight binding, potent and highly selective inhibitor of inducible nitric oxide synthase (Kd = 7 nM). Selective over nNOS and eNOS (Ki values are 2 and 50 ??M respectively). Cell-permeable and active in vivo.

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1400W Dihydrochloride 5mg 5mg  | Purity Not Available

Adooq Bioscience

1400W Dihydrochloride is a slow, tight binding, potent and highly selective inhibitor of inducible nitric oxide synthase (Kd = 7 nM). Selective over nNOS and eNOS (Ki values are 2 and 50 ??M respectively). Cell-permeable and active in vivo.

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17-AAG 100mg 100mg  | Purity Not Available

Adooq Bioscience

17-AAG is an ansamycin antibiotic which acts as an anti-tumor agent. Specifically, 17-AAG binds and inhibits Hsp90.

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17-AAG 10mg 10mg  | Purity Not Available

Adooq Bioscience

17-AAG is an ansamycin antibiotic which acts as an anti-tumor agent. Specifically, 17-AAG binds and inhibits Hsp90.

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17-AAG 200mg 200mg  | Purity Not Available

Adooq Bioscience

17-AAG is an ansamycin antibiotic which acts as an anti-tumor agent. Specifically, 17-AAG binds and inhibits Hsp90.

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17-AAG 50mg 50mg  | Purity Not Available

Adooq Bioscience

17-AAG is an ansamycin antibiotic which acts as an anti-tumor agent. Specifically, 17-AAG binds and inhibits Hsp90.

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17-DMAG HCl (Alvespimycin) 10mM * 1mL in DMSO 10mM * 1mL in DMSO  | Purity Not Available

Adooq Bioscience

17-DMAG is a water-soluble analog of 17-AAG and geldanamycin that binds the ATP binding site of Hsp90 and inhibits its chaperone activity. Displays more potent antitumor activity than 17-AAG.

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17-DMAG HCl 100mg 100mg  | Purity Not Available

Adooq Bioscience

17-DMAG is a water-soluble analog of 17-AAG and geldanamycin that binds the ATP binding site of Hsp90 and inhibits its chaperone activity. Displays more potent antitumor activity than 17-AAG.

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17-DMAG HCl 200mg 200mg  | Purity Not Available

Adooq Bioscience

17-DMAG is a water-soluble analog of 17-AAG and geldanamycin that binds the ATP binding site of Hsp90 and inhibits its chaperone activity. Displays more potent antitumor activity than 17-AAG.

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17-DMAG HCl 25mg 25mg  | Purity Not Available

Adooq Bioscience

17-DMAG is a water-soluble analog of 17-AAG and geldanamycin that binds the ATP binding site of Hsp90 and inhibits its chaperone activity. Displays more potent antitumor activity than 17-AAG.

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17-DMAG HCl 50mg 50mg  | Purity Not Available

Adooq Bioscience

17-DMAG is a water-soluble analog of 17-AAG and geldanamycin that binds the ATP binding site of Hsp90 and inhibits its chaperone activity. Displays more potent antitumor activity than 17-AAG.

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1alpha-Hydroxy VD4 1mg 1mg  | Purity Not Available

Adooq Bioscience

1alpha-Hydroxy VD4 , a 1alpha(OH)D derivative, can effectively induce the differentiation of monoblastic leukaemia U937, P39/TSU and P31/FUJ cells.

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1alpha-Hydroxy VD4 5mg 5mg  | Purity Not Available

Adooq Bioscience

1alpha-Hydroxy VD4 , a 1alpha(OH)D derivative, can effectively induce the differentiation of monoblastic leukaemia U937, P39/TSU and P31/FUJ cells.

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1Mps1-IN-1 10mg 10mg  | Purity Not Available

Adooq Bioscience

Mps1-IN-1 is a highly potent and selectibe Mpsl inhibitor with IC50 of 367 nM; >1000-fold selectivity relative to the 352 member kinase panel with the major exceptions of Alk and Ltk.

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1Mps1-IN-1 25mg 25mg  | Purity Not Available

Adooq Bioscience

Mps1-IN-1 is a highly potent and selectibe Mpsl inhibitor with IC50 of 367 nM; >1000-fold selectivity relative to the 352 member kinase panel with the major exceptions of Alk and Ltk.

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1Mps1-IN-1 5mg 5mg  | Purity Not Available

Adooq Bioscience

Mps1-IN-1 is a highly potent and selectibe Mpsl inhibitor with IC50 of 367 nM; >1000-fold selectivity relative to the 352 member kinase panel with the major exceptions of Alk and Ltk.

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2-HG (sodium salt) 100mg 100mg  | Purity Not Available

Adooq Bioscience

a-Hydroxyglutaric acid (2-HG) is an a-hydroxy acid. It is normally metabolized to 2-oxoglutarate by D- and L-2-hydroxyglutarate dehydrogenases, and mutations in these enzymes cause 2-hydroxyglutaric aciduria, a neurometabolic disorder.

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2-HG (sodium salt) 500mg 500mg  | Purity Not Available

Adooq Bioscience

a-Hydroxyglutaric acid (2-HG) is an a-hydroxy acid. It is normally metabolized to 2-oxoglutarate by D- and L-2-hydroxyglutarate dehydrogenases, and mutations in these enzymes cause 2-hydroxyglutaric aciduria, a neurometabolic disorder.

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2-Methoxyestradiol 100mg 100mg  | Purity Not Available

Adooq Bioscience

2-Methoxyestradiol is an apoptotic, antiproliferative and antiangiogenic agent. Induces p53-induced apoptosis via two pathways: activation of p38 and NF-??B; and activation of JNK and AP-1 leading to Bcl-2 phosphorylation.

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2-Methoxyestradiol 10mg 10mg  | Purity Not Available

Adooq Bioscience

2-Methoxyestradiol is an apoptotic, antiproliferative and antiangiogenic agent. Induces p53-induced apoptosis via two pathways: activation of p38 and NF-??B; and activation of JNK and AP-1 leading to Bcl-2 phosphorylation.

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2-Methoxyestradiol 10mM * 1mL in DMSO 10mM * 1mL in DMSO  | Purity Not Available

Adooq Bioscience

2-Methoxyestradiol is an apoptotic, antiproliferative and antiangiogenic agent. Induces p53-induced apoptosis via two pathways: activation of p38 and NF-??B; and activation of JNK and AP-1 leading to Bcl-2 phosphorylation.

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2-Methoxyestradiol 200mg 200mg  | Purity Not Available

Adooq Bioscience

2-Methoxyestradiol is an apoptotic, antiproliferative and antiangiogenic agent. Induces p53-induced apoptosis via two pathways: activation of p38 and NF-??B; and activation of JNK and AP-1 leading to Bcl-2 phosphorylation.

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2-Methoxyestradiol 50mg 50mg  | Purity Not Available

Adooq Bioscience

2-Methoxyestradiol is an apoptotic, antiproliferative and antiangiogenic agent. Induces p53-induced apoptosis via two pathways: activation of p38 and NF-??B; and activation of JNK and AP-1 leading to Bcl-2 phosphorylation.

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