CSNpharm
CSNpharm
ONPG
1g
| ≥99%
CSNpharm
ONPG
5g
| ≥99%
CSNpharm
CSNpharm
Ophiopogonanone A, a natural product isolated and purified from the tubers of Ophiopogon japonicus(Thunb) Ker-Gawl., has oxygen free radicals(OFRs) scavenging effects, and can scavenge hydroxyl radical(·OH) and hydrogen peroxide(H2O2) in vitro.
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Ophiopogonin D, a natural product isolated and purified from Radix Ophiopogon japonicus, protects the heart against doxorubicin-induced autophagic injury by reduction of both ROS generation and the disruption of the mitochondrial membrane damage.
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Ophiopogonin D, a natural product isolated and purified from Radix Ophiopogon japonicus, protects the heart against doxorubicin-induced autophagic injury by reduction of both ROS generation and the disruption of the mitochondrial membrane damage.
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Ophiopogonin D, a natural product isolated and purified from Radix Ophiopogon japonicus, protects the heart against doxorubicin-induced autophagic injury by reduction of both ROS generation and the disruption of the mitochondrial membrane damage.
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Orcinol glucoside is anxiolytic agent without sedative effect. Orcinol glucoside improves depressive behaviour in CUMS rats by downregulating HPA axis hyperactivity and increasing BDNF expression and ERK1/2 phosphorylation in the hippocampus.
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Org 25543 HCl is a potent and selective glycine transporter type 2 (GlyT2) inhibitor with IC50 of 16 nM for hGlyT2.
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Org 25543 HCl is a potent and selective glycine transporter type 2 (GlyT2) inhibitor with IC50 of 16 nM for hGlyT2.
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CSNpharm
Org 25543 HCl is a potent and selective glycine transporter type 2 (GlyT2) inhibitor with IC50 of 16 nM for hGlyT2.
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CSNpharm
Org 25543 HCl is a potent and selective glycine transporter type 2 (GlyT2) inhibitor with IC50 of 16 nM for hGlyT2.
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Org-12962 is a potent, selective and orally active 5-HT2C receptor agonist with a pEC50 value of 7.01. Org-12962 also exhibits high effacy for the 5-HT2A and 5-HT2B receptor with pEC50s of 6.38 and 6.28, respectively.Org-12962 displays antiaversive effects in a rat model of panic-like anxiety.
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Orientin, a natural product isolated and purified from the herbs of Polygonum orientale Linn., is a candidate therapeutic agent for treatment of vascular inflammatory diseases via inhibition of the HMGB1 signaling pathway, protects vascular barrier integrity by inhibiting hyperpermeability, expression of CAMs, and adhesion and migration of leukocytes, thereby endorsing its usefulness as a therapy […]
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Orientin, a natural product isolated and purified from the herbs of Polygonum orientale Linn., is a candidate therapeutic agent for treatment of vascular inflammatory diseases via inhibition of the HMGB1 signaling pathway, protects vascular barrier integrity by inhibiting hyperpermeability, expression of CAMs, and adhesion and migration of leukocytes, thereby endorsing its usefulness as a therapy […]
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Orientin, a natural product isolated and purified from the herbs of Polygonum orientale Linn., is a candidate therapeutic agent for treatment of vascular inflammatory diseases via inhibition of the HMGB1 signaling pathway, protects vascular barrier integrity by inhibiting hyperpermeability, expression of CAMs, and adhesion and migration of leukocytes, thereby endorsing its usefulness as a therapy […]
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CSNpharm
Orientin, a natural product isolated and purified from the herbs of Polygonum orientale Linn., is a candidate therapeutic agent for treatment of vascular inflammatory diseases via inhibition of the HMGB1 signaling pathway, protects vascular barrier integrity by inhibiting hyperpermeability, expression of CAMs, and adhesion and migration of leukocytes, thereby endorsing its usefulness as a therapy […]
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Ormeloxifene, an estrogen receptor regulator, suppresses prostate tumor growth and metastatic phenotypes via inhibition of oncogenic β-catenin signaling and EMT progression
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Ormeloxifene, an estrogen receptor regulator, suppresses prostate tumor growth and metastatic phenotypes via inhibition of oncogenic β-catenin signaling and EMT progression
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Ormeloxifene, an estrogen receptor regulator, suppresses prostate tumor growth and metastatic phenotypes via inhibition of oncogenic β-catenin signaling and EMT progression
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Ornidazole is an antibiotic used to treat some protozoan infections and investigated for use in Crohn’s disease after bowel resection.
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Ornidazole is an antibiotic used to treat some protozoan infections and investigated for use in Crohn’s disease after bowel resection.
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Orobol, a natural product isolated and purified from the herb of Wisteria sinensis, has sensitization effect may be by mitochondrial alterations in these cells, produces cisplatin sensitivity in human ovarian carcinoma cells by inducing apoptosis through the MT-dependent signaling pathway, and regulate PX sensitivity by reciprocally altering the proportion of tubulin isotype expression and PX-induced […]
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Orobol, a natural product isolated and purified from the herb of Wisteria sinensis, has sensitization effect may be by mitochondrial alterations in these cells, produces cisplatin sensitivity in human ovarian carcinoma cells by inducing apoptosis through the MT-dependent signaling pathway, and regulate PX sensitivity by reciprocally altering the proportion of tubulin isotype expression and PX-induced […]
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Oroxin A is the major component of an ethanol-water Oroxylum indicum (L.) Kurz (Bignoniaceae) seed extract (OISE), activates peroxisome proliferator-activated receptor γ (PPARγ) by docking into the PPARγ protein ligand-binding domain. Oroxin A exhibits an inhibitory activity against α-glucosidase and an antioxidant capacity[1]. Oroxin A exerts anti-breast cancer effects by inducing ER stress-mediated senescence[2].
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Oroxin A is the major component of an ethanol-water Oroxylum indicum (L.) Kurz (Bignoniaceae) seed extract (OISE), activates peroxisome proliferator-activated receptor γ (PPARγ) by docking into the PPARγ protein ligand-binding domain. Oroxin A exhibits an inhibitory activity against α-glucosidase and an antioxidant capacity[1]. Oroxin A exerts anti-breast cancer effects by inducing ER stress-mediated senescence[2].
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Oroxin A is the major component of an ethanol-water Oroxylum indicum (L.) Kurz (Bignoniaceae) seed extract (OISE), activates peroxisome proliferator-activated receptor γ (PPARγ) by docking into the PPARγ protein ligand-binding domain. Oroxin A exhibits an inhibitory activity against α-glucosidase and an antioxidant capacity[1]. Oroxin A exerts anti-breast cancer effects by inducing ER stress-mediated senescence[2].
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Oroxylin A shows inhibition of the dopamine reuptake and allosteric modulation of GABAA receptor. It is a natural active flavonoid isolated from scutellaria baicalensis and oroxylum indicum.
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Oroxylin A shows inhibition of the dopamine reuptake and allosteric modulation of GABAA receptor. It is a natural active flavonoid isolated from scutellaria baicalensis and oroxylum indicum.
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Oroxylin A shows inhibition of the dopamine reuptake and allosteric modulation of GABAA receptor. It is a natural active flavonoid isolated from scutellaria baicalensis and oroxylum indicum.
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Orphenadrine Citrate is both an uncompetitive NMDA receptor antagonist IC50 value of 16.2 µM and a mAChR antagonist with IC50 value of 0.37 µM.
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Orphenadrine Citrate is both an uncompetitive NMDA receptor antagonist IC50 value of 16.2 µM and a mAChR antagonist with IC50 value of 0.37 µM.
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Orsellinic acid, a natural product isolated and purified from the Chaetomium globosum endophytic, can block platelet activating factor (PAF)-mediated neuronal apoptosis without affecting G-protein coupled receptor (PAFR)-mediated neuroprotection, it can effectively attenuate PAFR-independent neuronal apoptosis.
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Orsellinic acid, a natural product isolated and purified from the Chaetomium globosum endophytic, can block platelet activating factor (PAF)-mediated neuronal apoptosis without affecting G-protein coupled receptor (PAFR)-mediated neuroprotection, it can effectively attenuate PAFR-independent neuronal apoptosis.
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CSNpharm
Orsellinic acid, a natural product isolated and purified from the Chaetomium globosum endophytic, can block platelet activating factor (PAF)-mediated neuronal apoptosis without affecting G-protein coupled receptor (PAFR)-mediated neuroprotection, it can effectively attenuate PAFR-independent neuronal apoptosis.
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